principiascientificinternational--2020-05-29--Study Shows Climate Change Expands Migratory Bird Ranges; Media Freaks

A newly published study shows North American birds are taking advantage of global warming to expanding their ranges northward , without any shrinkage in the southern edge of their North American ranges .
Rather than celebrate this good news for birds , climate alarmists and their media puppets are crying “ Crisis !
” A Google News search this morning for the term “ climate change ” shows articles about the new bird-range study are among the top search results .
Incredibly , the titles for media articles about the study include , “ Migratory Birds Are Failing to Adapt @ @ @ @ @ @ @ birds in the Eastern US are struggling to adapt to climate change , ” and “ National Audubon Society Says Climate Change Is Pushing Bird Boundaries , Community Scientists Confirm.
” Saying birds are “ struggling ” and “ failing ” to adapt to climate change , or that climate change is “ pushing ” bird boundaries , are grossly misleading ways to describe the good news of expanding bird ranges .
In the study , wildlife researchers working for the federal government tracked bird ranges during the past 50 years .
They @ @ @ @ @ @ @ the National Academy of Sciences .
• Annual resident bird species ( birds that do not migrate ) have , as a whole , increased their ranges northward toward the poles without losing any of their southern ranges .
That is wonderful news .
• Birds that migrate within North America have increased their ranges northward toward the poles without losing any of their southern ranges .
That is also wonderful news .
• Birds that migrate to North America from the tropics have had no change in their northern ranges , though @ @ @ @ @ @ @ nations appears to be shrinking .
That is not good news .
So , most North American birds have larger ranges today , thanks to global warming .
Annual resident bird species and migratory birds within North America have not experienced shrinking southern ranges .
The only concerning part of the survey is the subset of birds that winter outside the United States in Third World countries .
They appear to have shrinking ranges within those Third World countries .
Overall , the findings are quite good news for birds and should be @ @ @ @ @ @ @ subset of the study that might raise concern , declining ranges in Third World nations , is quite a stretch to blame on global warming .
If all other bird species , with southern ranges not in Third World nations , see no decline in their southern ranges , why would there be shrinkage of southern ranges only in Third World countries ?
The answer can likely be found in what the authors of the study explicitly acknowledge – “ [ T ] he primary threats to North American birds are thought to @ @ @ @ @ @ @ and direct and indirect anthropogenic mortality.
” Also , “ these threats are likely the primary drivers of declines in North America ’ s ” birds , the authors reported .
Habitat loss and other threats to bird species are much greater in tropical Third World countries than in eco-conscious North America .
The North American bird range is growing .
Birds that spend some of their time in North America and some of their time in Third World nations experience no range shrinkage in their North American ranges but some shrinkage in @ @ @ @ @ @ @ cause for the shrinkage of southern ranges for birds wintering in Third World countries clearly appears to be non-climate pressures on birds and other species in Third World countries .
Indeed , the authors themselves note “ deforestation and other factors in tropical nations ” may be pressuring that subset of birds migrating from tropical nations .
In summary , the new study on climate change and bird ranges is good news .
As a general rule , global warming is causing an expansion of bird ranges .
To the extent a subset @ @ @ @ @ @ @ , the reason appears to be non-climate pressures in Third World countries .
When the media describe the overall good news from the study as birds “ struggling , ” “ failing , ” or having their boundaries “ pushed ” by global warming , it reveals their biased and dishonest agenda .
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Study Shows Climate Change Expands Migratory Bird Ranges; Media Freaks
Written by James Taylor
A newly published study shows North American birds are taking advantage of global warming to expanding their ranges northward, without any shrinkage in the southern edge of their North American ranges.
Rather than celebrate this good news for birds, climate alarmists and their media puppets are crying “Crisis!”
A Google News search this morning for the term “climate change” shows articles about the new bird-range study are among the top search results.
Incredibly, the titles for media articles about the study include, “Migratory Birds Are Failing to Adapt to Climate Change,” “Migratory birds in the Eastern US are struggling to adapt to climate change,” and “National Audubon Society Says Climate Change Is Pushing Bird Boundaries, Community Scientists Confirm.”
Saying birds are “struggling” and “failing” to adapt to climate change, or that climate change is “pushing” bird boundaries, are grossly misleading ways to describe the good news of expanding bird ranges.
In the study, wildlife researchers working for the federal government tracked bird ranges during the past 50 years. They published their results in the Proceedings of the National Academy of Sciences.
The researchers found:
Annual resident bird species (birds that do not migrate) have, as a whole, increased their ranges northward toward the poles without losing any of their southern ranges. That is wonderful news.
Birds that migrate within North America have increased their ranges northward toward the poles without losing any of their southern ranges. That is also wonderful news.
Birds that migrate to North America from the tropics have had no change in their northern ranges, though their southern range in Third World tropical nations appears to be shrinking. That is not good news.
So, most North American birds have larger ranges today, thanks to global warming. Annual resident bird species and migratory birds within North America have not experienced shrinking southern ranges.
The only concerning part of the survey is the subset of birds that winter outside the United States in Third World countries.
They appear to have shrinking ranges within those Third World countries. Overall, the findings are quite good news for birds and should be reported as such.
Even the one subset of the study that might raise concern, declining ranges in Third World nations, is quite a stretch to blame on global warming.
If all other bird species, with southern ranges not in Third World nations, see no decline in their southern ranges, why would there be shrinkage of southern ranges only in Third World countries?
The answer can likely be found in what the authors of the study explicitly acknowledge – “[T]he primary threats to North American birds are thought to include habitat loss, invasive species, and direct and indirect anthropogenic mortality.”
Also, “these threats are likely the primary drivers of declines in North America’s” birds, the authors reported.
Habitat loss and other threats to bird species are much greater in tropical Third World countries than in eco-conscious North America.
The North American bird range is growing. Birds that spend some of their time in North America and some of their time in Third World nations experience no range shrinkage in their North American ranges but some shrinkage in their Third World ranges.
The driving cause for the shrinkage of southern ranges for birds wintering in Third World countries clearly appears to be non-climate pressures on birds and other species in Third World countries.
Indeed, the authors themselves note “deforestation and other factors in tropical nations” may be pressuring that subset of birds migrating from tropical nations.
In summary, the new study on climate change and bird ranges is good news. As a general rule, global warming is causing an expansion of bird ranges.
To the extent a subset of bird species defies the overall trend, the reason appears to be non-climate pressures in Third World countries.
When the media describe the overall good news from the study as birds “struggling,” “failing,” or having their boundaries “pushed” by global warming, it reveals their biased and dishonest agenda.
Read more at Climate Realism
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principiascientificinternational--2020-05-29--‘Dubious’ Record-High Temp Kept Despite Known Problems

‘ Dubious ’ Record-High Temp Kept Despite Known Problems Leading German daily Bild here reports on the controversy , which still continues to swirl , over Germany ’ s all-time record-high temperature recorded last year in North Germany near the Dutch border ( pictured ) .
Independent meteorologists say the readings need to be thrown out .
Last year on July 25th , the Lingen thermometer reached a whopping 42.6°C , far eclipsing the old German all-time record of 40.3°C .
But that recording quickly came under fire by independent weather experts who say the station data was corrupted by @ @ @ @ @ @ @ located in a depression in the earth , near a parking lot , and shielded by trees from the wind , thus creating the ideal conditions for trapping heat .
Last year NTZ reported on the controversial record here noting that surrounding stations did not even come close to record reading in Lingen .
What follows is a comparison of the Lingen ’ s readings to those of 6 nearby stations over the five day period , July 23 – July 27 : On every single day , Lingen handily beat its neighbors @ @ @ @ @ @ @ .
Today , a year later – as reported by Bild – meteorologists continue to slam the Lingen record .
Bild writes that the Lingen station today “ keeps on recording record-breaking temperatures : Bild also notes that German meteorologist Dominik Jung ( wetternet.de ) “ massively doubts the results of the station in Lingen ” and that “ it is dubious that the DWD allows such a weather station at all.
” Bild also cites meteorologist Dr. Karsten Brandt of donnerwetter.de : “ The station in Lingen has the problem that it @ @ @ @ @ @ @ heats up the temperatures .
This is definitely not the warmest place in Germany.
” So is Germany ’ s DWD National Weather Service , which operates the station , ready to dump Lingen ’ s overheated data ?
Bild reports that the DWD plans to stick to the Lingen readings , deeming them to be of good scientific quality .
The DWD replied to Bild : “ As a result of a site assessment , it can be stated that the station conditions have no serious influence on the temperature measurements and @ @ @ @ @ @ @ ” Exactly .
There ’ s nothing suspicious at all about a station routinely being 3°C warmer than all the others around it .
Silly anyone would get the idea to question that .
Read more at No Tricks Zone PRINCIPIA SCIENTIFIC INTERNATIONAL , legally registered in the UK as a company incorporated for charitable purposes .
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‘Dubious’ Record-High Temp Kept Despite Known Problems
Written by Pierre Gosselin
Leading German daily Bild here reports on the controversy, which still continues to swirl, over Germany’s all-time record-high temperature recorded last year in North Germany near the Dutch border (pictured).
Independent meteorologists say the readings need to be thrown out.
Last year on July 25th, the Lingen thermometer reached a whopping 42.6°C, far eclipsing the old German all-time record of 40.3°C.
But that recording quickly came under fire by independent weather experts who say the station data was corrupted by siting issues.
The Lingen station is located in a depression in the earth, near a parking lot, and shielded by trees from the wind, thus creating the ideal conditions for trapping heat.
Comparison to nearby stations shows a huge anomaly
Last year NTZ reported on the controversial record here noting that surrounding stations did not even come close to record reading in Lingen.
What follows is a comparison of the Lingen’s readings to those of 6 nearby stations over the five day period, July 23 – July 27:
On every single day, Lingen handily beat its neighbors by at times huge, red-flag-raising margins.
Today, a year later – as reported by Bild – meteorologists continue to slam the Lingen record.
Almost 4°C too hot?
Bild writes that the Lingen station today “keeps on recording record-breaking temperatures:
On May 20th, the station had 23.6 degrees. In the neighborhood it was below 20 degrees. On May 10, 27.7 degrees were measured in Lingen, in the neighborhood it was barely over 24 degrees.”
“Massive doubts”… “definitely not the warmest place”
Bild also notes that German meteorologist Dominik Jung (wetternet.de) “massively doubts the results of the station in Lingen” and that “it is dubious that the DWD allows such a weather station at all.”
Jung adds: “The station should be moved.”
Bild also cites meteorologist Dr. Karsten Brandt of donnerwetter.de: “The station in Lingen has the problem that it is located in a depression, which heats up the temperatures. This is definitely not the warmest place in Germany.”
So is Germany’s DWD National Weather Service, which operates the station, ready to dump Lingen’s overheated data? Bild reports that the DWD plans to stick to the Lingen readings, deeming them to be of good scientific quality.
The DWD replied to Bild: “As a result of a site assessment, it can be stated that the station conditions have no serious influence on the temperature measurements and therefore do not contradict the WMO standards.”
Exactly. There’s nothing suspicious at all about a station routinely being 3°C warmer than all the others around it. Silly anyone would get the idea to question that.
Read more at No Tricks Zone
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principiascientificinternational--2020-05-31--Scientists Expose Lancet Study Critical of HCQ COVID-19 Treatment

A massive study that raised serious health concerns about hydroxychloroquine , the malaria drug President Donald Trump has reportedly taken as a coronavirus preventive , is now under scrutiny from more than 180 scientists worldwide who are asking the research team to release its data for outside analysis .
When the study was published last week in the Lancet , a high-profile medical journal , it drew widespread media attention , including from BuzzFeed News .
Its massive dataset — consisting of 96,000 hospitalized COVID-19 patients across six continents — seemed to offer the most definitive examination to date of @ @ @ @ @ @ @ coronavirus , and also linked it to a higher risk of death .
But the letter , which went online on Thursday , raises questions about some seemingly inconsistent data in the paper .
Among the scientists ’ 10 concerns are that the average daily doses of hydroxychloroquine were higher than the FDA-recommended amounts and that data reportedly from Australian patients did not seem to match data from the Australian government .
This week , the Guardian reported that it could not confirm with several of that country ’ s health agencies that @ @ @ @ @ @ @ The study ’ s authors , led by Mandeep Mehra of Harvard Medical School , have repeatedly declined to release their underlying data .
On Friday , the study ’ s research team corrected some of its data but said its conclusions remained the same .
Not only did the study find no evidence that the malaria drug effectively combats the coronavirus , but it also linked the drug to serious heart problems .
Several other studies have previously reached similar conclusions , and the FDA has acknowledged the cardiac risks of this @ @ @ @ @ @ @ warning that the drugs not be used outside of a hospital setting .
For the first time , the Lancet study also found a link with a higher rate of deaths , finding that hospitalized patients who were given hydroxychloroquine were at least 33 % more likely to die than those who did not receive the treatment .
In the wake of the Lancet study , two major hydroxychloroquine clinical trials — one by the World Health Organization , another in the United Kingdom — were put on pause .
And the governments @ @ @ @ @ @ @ banned the drug from being used as a coronavirus treatment .
“ Everything points to a drug that has no efficacy , ” said Eric Topol , a cardiologist at the Scripps Research Translational Institute , who hasn ’ t signed the letter but has expressed skepticism publicly about the use of hydroxychloroquine as a treatment .
“ There ’ s no sign that it helps anyone .
We know it has significant side effects that are worrisome , ” including cardiac arrest and a dangerously rapid heart rate called ventricular tachycardia .
@ @ @ @ @ @ @ the Lancet study that didn ’ t seem to add up .
One of the biggest concerns of the letter ’ s signatories was that the authors had not released their code or data , even though the Lancet has signed a pledge to share COVID-19–related data .
said James Watson , a Thailand-based statistician with the University of Oxford ’ s Centre for Tropical Medicine and Global Health , who led the drafting of the letter , in an email to BuzzFeed News .
In response to the correction issued on Friday @ @ @ @ @ @ @ have not addressed the other nine points referred to in the letter.
” According to the Lancet study , the patient data came from electronic health records , supply chain databases , and financial records .
It was collected by Surgisphere , a Chicago-based health data analytics company led by Sapan Desai , one of the study ’ s coauthors .
Surgisphere says its data use agreements prevent it from sharing individual patient data and the names of its hospital customers , though it can conduct analyses and share aggregate findings .
“ @ @ @ @ @ @ @ reason that hospitals trust Surgisphere and we have been able to collect data from over 1,200 institutions across 46 countries , ” Desai told BuzzFeed News by email .
But the signatories on the letter say the researchers should at least share aggregated patient data at the hospital level .
They are also asking for an independent analysis and for the Lancet to release the peer review comments made about the study prior to publication .
Topol , the cardiologist at Scripps , said he had never heard of Surgisphere before the study @ @ @ @ @ @ @ ’ t done is release the data for others to analyze , ” he said .
“ That is important , and I think they should do that.
” In the correction issued Friday , the study ’ s authors fixed the numbers of participants from Australia and Asia .
One hospital tagged as belonging to the Australia region should have instead been assigned to the Asia region , according to the notice .
Other corrections were also issued , but the notice stated that none of the changes altered the paper ’ @ @ @ @ @ @ @ said the journal will soon publish responses to the study and a response from the authors .
Mehra , the study ’ s lead author , said he stands by the research .
He and his team used Surgisphere ’ s data “ in the absence of a large , robust and publicly available dataset on hydroxychloroquine or chloroquine , and the lack of scientific evidence regarding the safety and benefits of these treatments for hospitalized Covid-19 patients , ” said Mehra , medical director of the Brigham and Women ’ s Hospital @ @ @ @ @ @ @ of medicine at Harvard Medical School , through a spokesperson .
Mehra , Desai , and other researchers also used Surgisphere data in a study published this month in the New England Journal of Medicine about cardiovascular disease in COVID-19 patients .
Watson , the researcher organizing the letter , has criticized some of that paper ’ s supplementary data about age and mortality rates .
Mehra and Desai did not answer questions about this additional study .
Jennifer Zeis , a New England Journal of Medicine spokesperson , said that the publication @ @ @ @ @ @ @ PRINCIPIA SCIENTIFIC INTERNATIONAL , legally registered in the UK as a company incorporated for charitable purposes .
Head Office : 27 Old Gloucester Street , London WC1N 3AX .
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Scientists Expose Lancet Study Critical of HCQ COVID-19 Treatment
Written by Stephanie M Lee
A massive study that raised serious health concerns about hydroxychloroquine, the malaria drug President Donald Trump has reportedly taken as a coronavirus preventive, is now under scrutiny from more than 180 scientists worldwide who are asking the research team to release its data for outside analysis.
When the study was published last week in the Lancet, a high-profile medical journal, it drew widespread media attention, including from BuzzFeed News. Its massive dataset — consisting of 96,000 hospitalized COVID-19 patients across six continents — seemed to offer the most definitive examination to date of hydroxychloroquine’s inability to fight the coronavirus, and also linked it to a higher risk of death.
But the letter, which went online on Thursday, raises questions about some seemingly inconsistent data in the paper. Among the scientists’ 10 concerns are that the average daily doses of hydroxychloroquine were higher than the FDA-recommended amounts and that data reportedly from Australian patients did not seem to match data from the Australian government.
This week, the Guardian reported that it could not confirm with several of that country’s health agencies that they provided data to the study.
The study’s authors, led by Mandeep Mehra of Harvard Medical School, have repeatedly declined to release their underlying data.
On Friday, the study’s research team corrected some of its data but said its conclusions remained the same.
Not only did the study find no evidence that the malaria drug effectively combats the coronavirus, but it also linked the drug to serious heart problems. Several other studies have previously reached similar conclusions, and the FDA has acknowledged the cardiac risks of this and a related drug, chloroquine, warning that the drugs not be used outside of a hospital setting.
For the first time, the Lancet study also found a link with a higher rate of deaths, finding that hospitalized patients who were given hydroxychloroquine were at least 33{154653b9ea5f83bbbf00f55de12e21cba2da5b4b158a426ee0e27ae0c1b44117} more likely to die than those who did not receive the treatment.
In the wake of the Lancet study, two major hydroxychloroquine clinical trials — one by the World Health Organization, another in the United Kingdom — were put on pause. And the governments of France, Belgium, and Italy banned the drug from being used as a coronavirus treatment.
“Everything points to a drug that has no efficacy,” said Eric Topol, a cardiologist at the Scripps Research Translational Institute, who hasn’t signed the letter but has expressed skepticism publicly about the use of hydroxychloroquine as a treatment. “There’s no sign that it helps anyone. We know it has significant side effects that are worrisome,” including cardiac arrest and a dangerously rapid heart rate called ventricular tachycardia.
Even so, scientists found aspects of the Lancet study that didn’t seem to add up.
One of the biggest concerns of the letter’s signatories was that the authors had not released their code or data, even though the Lancet has signed a pledge to share COVID-19–related data.
“Many of us in the scientific community were just very angry at seeing a poorly written and executed study published in The Lancet, given loads of publicity, and then having a hugely negative impact on carefully planned clinical trials around the world,”
said James Watson, a Thailand-based statistician with the University of Oxford’s Centre for Tropical Medicine and Global Health, who led the drafting of the letter, in an email to BuzzFeed News.
In response to the correction issued on Friday, Watson said, “The authors have not addressed the other nine points referred to in the letter.”
According to the Lancet study, the patient data came from electronic health records, supply chain databases, and financial records. It was collected by Surgisphere, a Chicago-based health data analytics company led by Sapan Desai, one of the study’s coauthors.
Surgisphere says its data use agreements prevent it from sharing individual patient data and the names of its hospital customers, though it can conduct analyses and share aggregate findings. “Our strong privacy standards are a major reason that hospitals trust Surgisphere and we have been able to collect data from over 1,200 institutions across 46 countries,” Desai told BuzzFeed News by email.
But the signatories on the letter say the researchers should at least share aggregated patient data at the hospital level. They are also asking for an independent analysis and for the Lancet to release the peer review comments made about the study prior to publication.
Topol, the cardiologist at Scripps, said he had never heard of Surgisphere before the study. “The main thing they haven’t done is release the data for others to analyze,” he said. “That is important, and I think they should do that.”
In the correction issued Friday, the study’s authors fixed the numbers of participants from Australia and Asia. One hospital tagged as belonging to the Australia region should have instead been assigned to the Asia region, according to the notice. Other corrections were also issued, but the notice stated that none of the changes altered the paper’s conclusions.
Lancet spokesperson Jessica Kleyn said the journal will soon publish responses to the study and a response from the authors.
Mehra, the study’s lead author, said he stands by the research. He and his team used Surgisphere’s data “in the absence of a large, robust and publicly available dataset on hydroxychloroquine or chloroquine, and the lack of scientific evidence regarding the safety and benefits of these treatments for hospitalized Covid-19 patients,” said Mehra, medical director of the Brigham and Women’s Hospital Heart and Vascular Center and a professor of medicine at Harvard Medical School, through a spokesperson.
Mehra, Desai, and other researchers also used Surgisphere data in a study published this month in the New England Journal of Medicine about cardiovascular disease in COVID-19 patients. Watson, the researcher organizing the letter, has criticized some of that paper’s supplementary data about age and mortality rates.
Mehra and Desai did not answer questions about this additional study. Jennifer Zeis, a New England Journal of Medicine spokesperson, said that the publication was looking into the questions raised.
Read more at www.buzzfeednews.com
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principiascientificinternational--2020-05-31--Top Medical Journal Exposes Myth of Face Masks for COVID-19

A new paper from the New England Journal of Medicine exposes the myth that mask wearing will protect you from the corona virus .
It warns that “ wearing a mask outside health care facilities offers little , if any , protection from infection.
” The paper , ‘ Universal Masking in Hospitals in the Covid-19 Era ‘ ( May 21 , 2020 ) suggests that advocating the wearing of masks in public merely offers a psychological , not a medical , reassurance .
[ 1 ] The paper is also careful to balance those statements by then explaining why @ @ @ @ @ @ @ is advisable and recommended .
However , the policies of compulsory mask wearing by large sections of the healthy population is a very different context from that found inside hospitals .
The authors explain : “ What is clear , however , is that universal masking alone is not a panacea .
A mask will not protect providers caring for a patient with active Covid-19 if it ’ s not accompanied by meticulous hand hygiene , eye protection , gloves , and a gown .
A mask alone will not prevent health care @ @ @ @ @ @ @ hands and spreading the virus to patients and colleagues .
Focusing on universal masking alone may , paradoxically , lead to more transmission of Covid-19 if it diverts attention from implementing more fundamental infection-control measures.
” The overriding message is that masks serve symbolic rather than clinical roles when deployed outside of a medical facility .
The paper ends by declaring : PRINCIPIA SCIENTIFIC INTERNATIONAL , legally registered in the UK as a company incorporated for charitable purposes .
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Top Medical Journal Exposes Myth of Face Masks for COVID-19
Written by John O'Sullivan
A new paper from the New England Journal of Medicine exposes the myth that mask wearing will protect you from the corona virus. It warns that “wearing a mask outside health care facilities offers little, if any, protection from infection.”
The paper, ‘Universal Masking in Hospitals in the Covid-19 Era‘ (May 21, 2020) suggests that advocating the wearing of masks in public merely offers a psychological, not a medical, reassurance. [1]
The authors write:
“In many cases, the desire for widespread masking is a reflexive reaction to anxiety over the pandemic.”
The paper is also careful to balance those statements by then explaining why wearing a mask in a clinical setting is advisable and recommended. However, the policies of compulsory mask wearing by large sections of the healthy population is a very different context from that found inside hospitals. The authors explain:
“What is clear, however, is that universal masking alone is not a panacea. A mask will not protect providers caring for a patient with active Covid-19 if it’s not accompanied by meticulous hand hygiene, eye protection, gloves, and a gown. A mask alone will not prevent health care workers with early Covid-19 from contaminating their hands and spreading the virus to patients and colleagues. Focusing on universal masking alone may, paradoxically, lead to more transmission of Covid-19 if it diverts attention from implementing more fundamental infection-control measures.”
The overriding message is that masks serve symbolic rather than clinical roles when deployed outside of a medical facility. The paper ends by declaring:
“Expanded masking protocols’ greatest contribution may be to reduce the transmission of anxiety, over and above whatever role they may play in reducing transmission of Covid-19.”
Reference:
[1] N Engl J Med 2020; 382:e63 (published May 21, 2020) DOI: 10.1056/NEJMp2006372
Read more at www.nejm.org
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principiascientificinternational--2020-06-01--Coronavirus in a Time of Chimeras

Coronavirus in a Time of Chimeras

As the global COVID-19 scourge appears to recede , questions remain over the source and morphology of a virus that had locked down two-thirds of humanity over the first half of 2020 .
It may take years to satisfactorily decipher this extraordinary episode in human history .
Nonetheless , the novel coronavirus was not germinated in a vacuum .
The type of research conducted at the Wuhan Institute of Virology had ominous analogues worldwide .
These included the quest for super intelligence and the development of interspecies hybrids or chimeras .
What began as a scientific mission to remedy congenital @ @ @ @ @ @ @ race to create designer babies , super soldiers and transhumans through the aid of biotechnology and Artificial Intelligence .
21st century eugenics is tacitly justified by the need to boost “ national competitiveness ” .
China leads the way here .
In one revealing instance alone , genome sequencing giant BGI Shenzhen had procured and sequenced the DNA of more than 2,000 people – mostly Americans – with IQ scores of at least 160 .
According to Stephen Hsu , a theoretical physicist from Michigan State University and scientific adviser to BGI : @ @ @ @ @ @ @ bio-Google ” that will collate the “ world ’ s biological information and make it universally accessible and useful ” .
From 2012 onwards , it began to collaborate with the Bill & Melinda Gates Foundation .
Scientific endeavours like these are based on the assumption that an assemblage of smart samples will help in the identification and transplantation of optimal bits of DNA into future generations .
It is not dissuaded by the nurture over nature debate , even after exhaustive studies have failed to establish genetic variants associated with intelligence .
@ @ @ @ @ @ @ by Robert Plomin , a behavioural geneticist at King ’ s College London , had probed over 350,000 variations in single DNA letters across the genomes of 7,900 children but found no prized variant .
Curiously , most of the smart samples procured by BGI Shenzhen were sourced from Plomin ’ s research activities .
Periodic setbacks will not deter the proponents of “ procreative beneficence ” who argue that it is a human duty to augment the genetic codes of future generations1 .
Failure to do so is couched in terms of @ @ @ @ @ @ @ abuse2 .
If this sounds eerily familiar , look no further than the worldview that once animated Nazi Germany .
The eugenic zeitgeist has gripped China in a big way .
Under its Maternal and Infant Health Care Law ( 1994 ) , foetuses with potential hereditary diseases or deformities are recommended for abortion .
At the rate Beijing is building its eugenic utopia , the definition of serious deformity may ultimately include a genetically-diagnosed lower IQ .
Instead of raising an eyebrow , the law precipitated a headlong rush to select “ @ @ @ @ @ @ @ genetic diagnosis ( PGD ) .
The idea behind PGD is to screen and identify the most promising embryos for implantation and birth .
Combined with CRISPR gene-editing tools , next generation Chinese citizens are expected to exhibit remarkably higher IQs – at least according to bioethicists who fret over a future marked by the “ genetic haves ” and “ genetic have-nots ” .
China already has three CRISPR-edited babies whose current fate remains unclear .
In the aftermath of the COVID-19 contagion , the availability of “ smart samples ” would @ @ @ @ @ @ @ with the vaccination agenda of the Rockefeller Foundation and Bill Gates .
Incidentally , Gates grew up in a household that was heavily invested in population control and eugenics .
( photo : Bill ’ s father , William Gates Sr , below with the Rockefellers ) Our smart societies may inevitably face the existential question of “ live-lets ” and “ live-nots ” down the line .
The orchestrated rebellion towards selective extinction , if it occurs , has a tragicomical public face : A 17-year-old Swede who unceasingly exhorts the world @ @ @ @ @ @ @ and “ listen to the experts ” but who has little time to listen to her own school teachers .
What can future designer babies contribute to society ?
For one thing , we will be missing individuals like Beethoven ( deaf ) ; Albert Einstein ( learning disability/late development ) ; John Nash ( schizophrenia ) ; Andrea Boccelli ( congenital glaucoma ) and Vincent van Gogh ( chronic depression/anxiety ) .
A future Stephen Hawkings ( motor neurone disease ) and Greta Thunberg ( Asperger ’ s Syndrome – allegedly ) @ @ @ @ @ @ @ It is now an inconvenience to consider intelligence as a result of peer interactions , human environment and personal adversity .
Mapping out the complex and sometimes unpredictable interplay between 100 trillion synaptic connections in a human brain may take centuries .
Genetic manipulation is implicitly regarded as the eugenic wormhole that will accelerate the emergence of a global smart society .
The late billionaire paedophile , Jeffrey Epstein , was a prominent proponent of this eugenics philosophy .
Epstein intended to breed a “ super race of humans with his DNA by @ @ @ @ @ @ @ , genetic engineering and artificial intelligence.
” Welcome to Lebensborn 2.0 and it is all about saving the environment and humanity .
For now !
Prominent scientists linked to Epstein ’ s transhumanist fantasies included “ molecular engineer George Church ; Murray Gell-Mann , the discoverer of the quark ; the evolutionary biologist Stephen Jay Gould ; the neurologist and author Oliver Sacks ; and the theoretical physicist Frank Wilczek.
” The late Stephen Hawking – who will ironically flunk the genetic pre-screenings of tomorrow – was another Epstein associate .
Are misanthropes @ @ @ @ @ @ @ national competitiveness is a tacitly growing obsession among major powers .
Its hyper-materialistic focus is encapsulated by Russian scientist Denis Rebrikov : “ It currently costs about a million rubles ( $ 15,500 at the time ) to genetically change an embryo—more than a lot of cars—but prices will fall with greater use…I can see the billboard now : ‘ You Choose : a Hyundai Solaris or a Super-Child ?
’ ” Will that be an energy-efficient , coronavirus-resistant super child who will instinctively lead a low carbon-emitting lifestyle ?
The road to @ @ @ @ @ @ @ .
But why stop at children ?
From genetically engineered horses in Argentina that are supposedly faster , stronger and better jumpers to super-dogs in China that are comprehensively superior to the average mutt , the DNA of the entire natural world may be slated for a revolutionary redesign in the future .
We however can not create a future generation of superhumans without being adept at recombining genetic sequences across species .
That is the logic guiding eugenicists .
As a result , a slew of chimeras or interspecies hybrids have been @ @ @ @ @ @ @ These include human-monkey hybrids , monkey-pig hybrids , human-rabbit hybrids and a host of other lab-manufactured monstrosities .
Chimeras are created when human embryonic stem cells are injected into embryos from another species .
The goal , for the time being , is to induce the growth of targeted human organs .
Those facing terminal illnesses will no longer have to worry about long organ waiting lists .
A less controversial approach to human organ replacement is 3D bioprinting or its 4D bioprinting iteration .
These techniques involve the “ printing ” of @ @ @ @ @ @ @ of a transplant recipient , thereby eliminating the odds of organ rejection .
But why stop at replacement organs when we can have replacement humans altogether ?
Future generations must think like Einsteins , be as nimble as leopards and possess owl-like night visions .
And , of course , be virus-resistant as well !
The manipulation of the human genome is the new “ grand response ” to the venerable set of “ grand challenges ” .
Thanks to globalization , China is the go-to place for such genetic tinkering as some @ @ @ @ @ @ @ the West .
Since 2014 , the Wuhan Institute of Virology was the recipient of a two-stage grant worth $ 7.2 million from the United States government for gain-of-function research into bat coronaviruses .
According to a Newsweek report : Many scientists have criticized gain of function research , which involves manipulating viruses in the lab to explore their potential for infecting humans , because it creates a risk of starting a pandemic from accidental release .
Such caution has not deterred a flurry of research into microbial gene manipulation .
It may @ @ @ @ @ @ @ codes at the substrate levels is fraught with risks , as any systems theory scholar can attest3 .
COVID-19 was therefore not a Black Swan event but likely an “ emergent ” 4 outcome arising from complex genomic interactions and human folly .
To solely blame China for the coronavirus pandemic therefore may be a tad unfair .
Just as China is the factory of the world for foreign corporations , it is also the genetic incubator for a variety of viruses and chimeras for foreign governments and foundations .
Even so , @ @ @ @ @ @ @ the Salk Institute in California .
Research into the world ’ s first human-mouse hybrid was largely a Japanese affair .
The Portuguese in the meantime had created a virus chimera .
The British , on their end , had spawned a human-cow hybrid embryo in 2008 – perhaps reflective of the bovine disposition of those who consume its mainstream media .
Clinically-speaking , such analogies are not wholly unwarranted .
It was in Britain where the game-changing Dolly the Sheep was cloned in 1996 .
The transition from sheep to sheeple may @ @ @ @ @ @ @ century Jurassic Park ride .
Before the advent of gene-editing tools and supercomputing , it would have taken hundreds of years to create a viable chimera .
The Genetics-Industrial Complex and contact tracing-type Panopticons constitute a new growth area for nearly-bankrupt Tech Titans5 .
Is it any wonder that the mainstream media and their Big Tech owners are furiously censoring contrarian expert views on COVID-19 ?
The dangers of genome editing were in fact included in the Worldwide Threat Assessment reports submitted to United States Congress in 2016 and 2017 .
They were @ @ @ @ @ @ @ 2018 and 2019 reports – just as such risks were on the rise .
Is it a coincidence that the nations most affected by COVID-19 are the very ones that had either promoted or encouraged a variety of genetic experimentations that are contrary to nature ?
By the time this crisis is over , independent researchers should superimpose the maps of “ genetic superpowers ” with nations having the highest COVID-19 fatality rates or the worst socioeconomic fallouts .
There may likely be a good degree of overlap as the figure below indicates @ @ @ @ @ @ @ been opened and more hideous chimeras may emerge during this decade .
It is quite an irony that a new generation of artificially-manufactured and cerebrally-deficient “ thought leaders ” , academics and activists are being groomed to promote “ global governance ” – a concept due for a portentous mission creep in tandem with the Second Great Depression .
What will be their future worth in a eugenic global society that is centrally-controlled by a digital panopticon6 ?
“ Designer babies ” and “ super humans ” may also render many humans redundant @ @ @ @ @ @ @ as “ live-nots ” in the not-so-distant future ?
1 Savulescu J .
( 2001 ) Procreative beneficence : why we should select the best children .
Bioethics .
2001 ; 15 ( 5-6 ) :413‐426 .
2 Hammond J .
( 2010 ) Genetic engineering to avoid genetic neglect : from chance to responsibility .
Bioethics .
2010 ; 24 ( 4 ) :160‐169 .
3 Kauffman , S. A .
( 1993 ) .
The Origins of Order : Self-Organization and Selection in Evolution .
New York : Oxford University Press @ @ @ @ @ @ @ .
Bubble to Panopticon : Dark Undercurrents of the Big Data Torrent .
Kybernetes , Vol .
49 No .
3 , pp .
1046-1060. https : //doi.org/10.1108/K-06-2019-0403 About the author : Dr Mathew Maavak is a regular commentator on geostrategic issues .
He holds a PhD in Policy Studies from the Universiti Teknologi Malaysia and a master ’ s degree in International Communications from the University of Leeds .
His supervisor and personal tutor was the legendary ( late ) Prof Philip M. Taylor who had devised information warfare strategies against the @ @ @ @ @ @ @ , legally registered in the UK as a company incorporated for charitable purposes .
Head Office : 27 Old Gloucester Street , London WC1N 3AX .
Please DONATE TODAY To Help Our Non-Profit Mission To Defend The Scientific Method .

Coronavirus in a Time of Chimeras
Written by Mathew Maavak PhD
As the global COVID-19 scourge appears to recede, questions remain over the source and morphology of a virus that had locked down two-thirds of humanity over the first half of 2020. It may take years to satisfactorily decipher this extraordinary episode in human history.
Nonetheless, the novel coronavirus was not germinated in a vacuum. The type of research conducted at the Wuhan Institute of Virology had ominous analogues worldwide. These included the quest for super intelligence and the development of interspecies hybrids or chimeras.
Genetically-Enhanced Competitiveness
What began as a scientific mission to remedy congenital defects has rapidly morphed into a global race to create designer babies, super soldiers and transhumans through the aid of biotechnology and Artificial Intelligence. 21st century eugenics is tacitly justified by the need to boost “national competitiveness”.
China leads the way here. In one revealing instance alone, genome sequencing giant BGI Shenzhen had procured and sequenced the DNA of more than 2,000 people – mostly Americans – with IQ scores of at least 160. According to Stephen Hsu, a theoretical physicist from Michigan State University and scientific adviser to BGI:
“An exceptional person gets you an order of magnitude more statistical power than if you took random people from the population…”
BGI Shenzhen intends to become a “bio-Google” that will collate the “world’s biological information and make it universally accessible and useful”. From 2012 onwards, it began to collaborate with the Bill & Melinda Gates Foundation.
Scientific endeavours like these are based on the assumption that an assemblage of smart samples will help in the identification and transplantation of optimal bits of DNA into future generations. It is not dissuaded by the nurture over nature debate, even after exhaustive studies have failed to establish genetic variants associated with intelligence.
For example, a 2010 study led by Robert Plomin, a behavioural geneticist at King’s College London, had probed over 350,000 variations in single DNA letters across the genomes of 7,900 children but found no prized variant. Curiously, most of the smart samples procured by BGI Shenzhen were sourced from Plomin’s research activities.
Periodic setbacks will not deter the proponents of “procreative beneficence” who argue that it is a human duty to augment the genetic codes of future generations1. Failure to do so is couched in terms of “genetic neglect” and even child abuse2. If this sounds eerily familiar, look no further than the worldview that once animated Nazi Germany.
The eugenic zeitgeist has gripped China in a big way. Under its Maternal and Infant Health Care Law (1994), foetuses with potential hereditary diseases or deformities are recommended for abortion. At the rate Beijing is building its eugenic utopia, the definition of serious deformity may ultimately include a genetically-diagnosed lower IQ.
Instead of raising an eyebrow, the law precipitated a headlong rush to select “intelligent” babies through methods like preimplantation genetic diagnosis (PGD). The idea behind PGD is to screen and identify the most promising embryos for implantation and birth.
Combined with CRISPR gene-editing tools, next generation Chinese citizens are expected to exhibit remarkably higher IQs – at least according to bioethicists who fret over a future marked by the “genetic haves” and “genetic have-nots”. China already has three CRISPR-edited babies whose current fate remains unclear.
In the aftermath of the COVID-19 contagion, the availability of “smart samples” would have increased exponentially and may dovetail nicely with the vaccination agenda of the Rockefeller Foundation and Bill Gates. Incidentally, Gates grew up in a household that was heavily invested in population control and eugenics. (photo: Bill’s father, William Gates Sr, below with the Rockefellers)
Our smart societies may inevitably face the existential question of “live-lets” and “live-nots” down the line. The orchestrated rebellion towards selective extinction, if it occurs, has a tragicomical public face: A 17-year-old Swede who unceasingly exhorts the world to “listen to the science” and “listen to the experts” but who has little time to listen to her own school teachers.
What can future designer babies contribute to society? For one thing, we will be missing individuals like Beethoven (deaf); Albert Einstein (learning disability/late development); John Nash (schizophrenia); Andrea Boccelli (congenital glaucoma) and Vincent van Gogh (chronic depression/anxiety). A future Stephen Hawkings (motor neurone disease) and Greta Thunberg (Asperger’s Syndrome – allegedly) may be genetically disqualified before birth.
It is now an inconvenience to consider intelligence as a result of peer interactions, human environment and personal adversity. Mapping out the complex and sometimes unpredictable interplay between 100 trillion synaptic connections in a human brain may take centuries. Genetic manipulation is implicitly regarded as the eugenic wormhole that will accelerate the emergence of a global smart society.
The late billionaire paedophile, Jeffrey Epstein, was a prominent proponent of this eugenics philosophy. Epstein intended to breed a “super race of humans with his DNA by impregnating women at his New Mexico ranch, genetic engineering and artificial intelligence.” Welcome to Lebensborn 2.0 and it is all about saving the environment and humanity. For now!
(Photo below: Bill Gates with pedophile, Jeffrey Epstein)
Prominent scientists linked to Epstein’s transhumanist fantasies included “molecular engineer George Church; Murray Gell-Mann, the discoverer of the quark; the evolutionary biologist Stephen Jay Gould; the neurologist and author Oliver Sacks; and the theoretical physicist Frank Wilczek.” The late Stephen Hawking – who will ironically flunk the genetic pre-screenings of tomorrow – was another Epstein associate. Are misanthropes in charge of humanity now?
Eugenics-driven national competitiveness is a tacitly growing obsession among major powers. Its hyper-materialistic focus is encapsulated by Russian scientist Denis Rebrikov:
“It currently costs about a million rubles ($15,500 at the time) to genetically change an embryo—more than a lot of cars—but prices will fall with greater use…I can see the billboard now: ‘You Choose: a Hyundai Solaris or a Super-Child?’”
Will that be an energy-efficient, coronavirus-resistant super child who will instinctively lead a low carbon-emitting lifestyle? The road to hell is indeed paved with fanciful intentions.
But why stop at children? From genetically engineered horses in Argentina that are supposedly faster, stronger and better jumpers to super-dogs in China that are comprehensively superior to the average mutt, the DNA of the entire natural world may be slated for a revolutionary redesign in the future.
Crouching Chimeras, Hideous Hybrids
We however cannot create a future generation of superhumans without being adept at recombining genetic sequences across species. That is the logic guiding eugenicists. As a result, a slew of chimeras or interspecies hybrids have been spawned with the aid of CRISPR. These include human-monkey hybrids, monkey-pig hybrids, human-rabbit hybrids and a host of other lab-manufactured monstrosities.
Chimeras are created when human embryonic stem cells are injected into embryos from another species. The goal, for the time being, is to induce the growth of targeted human organs. Those facing terminal illnesses will no longer have to worry about long organ waiting lists. A less controversial approach to human organ replacement is 3D bioprinting or its 4D bioprinting iteration. These techniques involve the “printing” of a replacement organ from the stem cells of a transplant recipient, thereby eliminating the odds of organ rejection.
But why stop at replacement organs when we can have replacement humans altogether? Future generations must think like Einsteins, be as nimble as leopards and possess owl-like night visions. And, of course, be virus-resistant as well!
The manipulation of the human genome is the new “grand response” to the venerable set of “grand challenges”. Thanks to globalization, China is the go-to place for such genetic tinkering as some of these undertakings are technically illegal in the West. Since 2014, the Wuhan Institute of Virology was the recipient of a two-stage grant worth $7.2 million from the United States government for gain-of-function research into bat coronaviruses. According to a Newsweek report:
Many scientists have criticized gain of function research, which involves manipulating viruses in the lab to explore their potential for infecting humans, because it creates a risk of starting a pandemic from accidental release.
Such caution has not deterred a flurry of research into microbial gene manipulation. It may have instead spawned COVID-19. Recombining genetic codes at the substrate levels is fraught with risks, as any systems theory scholar can attest3. COVID-19 was therefore not a Black Swan event but likely an “emergent”4 outcome arising from complex genomic interactions and human folly.
To solely blame China for the coronavirus pandemic therefore may be a tad unfair. Just as China is the factory of the world for foreign corporations, it is also the genetic incubator for a variety of viruses and chimeras for foreign governments and foundations. Even so, the human-pig chimera was the creation of the Salk Institute in California. Research into the world’s first human-mouse hybrid was largely a Japanese affair. The Portuguese in the meantime had created a virus chimera.
The British, on their end, had spawned a human-cow hybrid embryo in 2008 – perhaps reflective of the bovine disposition of those who consume its mainstream media. Clinically-speaking, such analogies are not wholly unwarranted. It was in Britain where the game-changing Dolly the Sheep was cloned in 1996. The transition from sheep to sheeple may turn out to be a short 21st century Jurassic Park ride.
Coincidences and Consequences
Before the advent of gene-editing tools and supercomputing, it would have taken hundreds of years to create a viable chimera. The Genetics-Industrial Complex and contact tracing-type Panopticons constitute a new growth area for nearly-bankrupt Tech Titans5. Is it any wonder that the mainstream media and their Big Tech owners are furiously censoring contrarian expert views on COVID-19?
The dangers of genome editing were in fact included in the Worldwide Threat Assessment reports submitted to United States Congress in 2016 and 2017. They were either omitted or glossed over in the 2018 and 2019 reports – just as such risks were on the rise.
Is it a coincidence that the nations most affected by COVID-19 are the very ones that had either promoted or encouraged a variety of genetic experimentations that are contrary to nature? By the time this crisis is over, independent researchers should superimpose the maps of “genetic superpowers” with nations having the highest COVID-19 fatality rates or the worst socioeconomic fallouts. There may likely be a good degree of overlap as the figure below indicates.
A Pandora’s Box has been opened and more hideous chimeras may emerge during this decade. It is quite an irony that a new generation of artificially-manufactured and cerebrally-deficient “thought leaders”, academics and activists are being groomed to promote “global governance” – a concept due for a portentous mission creep in tandem with the Second Great Depression. What will be their future worth in a eugenic global society that is centrally-controlled by a digital panopticon6?
“Designer babies” and “super humans” may also render many humans redundant. Will the genetic have-nots be reclassified as “live-nots” in the not-so-distant future?
References
1 Savulescu J. (2001) Procreative beneficence: why we should select the best children. Bioethics. 2001;15(5-6):413‐426.
2 Hammond J. (2010) Genetic engineering to avoid genetic neglect: from chance to responsibility. Bioethics. 2010;24(4):160‐169.
3 Kauffman, S. A. (1993). The Origins of Order: Self-Organization and Selection in Evolution. New York: Oxford University Press
4 O’Connor, Timothy; Wong, Hong Yu (2012). Edward N. Zalta (ed.). “Emergent Properties”. The Stanford Encyclopedia of Philosophy (Spring 2012 Edition).
5,6 Maavak, M. (2019). Bubble to Panopticon: Dark Undercurrents of the Big Data Torrent. Kybernetes, Vol. 49 No. 3, pp. 1046-1060. https://doi.org/10.1108/K-06-2019-0403
About the author: Dr Mathew Maavak is a regular commentator on geostrategic issues. He holds a PhD in Policy Studies from the Universiti Teknologi Malaysia and a master’s degree in International Communications from the University of Leeds. His supervisor and personal tutor was the legendary (late) Prof Philip M. Taylor who had devised information warfare strategies against the former Soviet Union.
PRINCIPIA SCIENTIFIC INTERNATIONAL, legally registered in the UK as a company incorporated for charitable purposes. Head Office: 27 Old Gloucester Street, London WC1N 3AX.
Please DONATE TODAY To Help Our Non-Profit Mission To Defend The Scientific Method.
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principiascientificinternational--2020-06-01--Does Aluminum Contamination Cause Alzheimer’s Disease?

There is speculation of a link between higher levels of aluminum ( aluminium ) in our bodies and an increased risk of Alzheimer ’ s Disease .
Note the American spelling of aluminium in these articles , which they pronounce ‘ aloominum ’ .
Below we cover some of the more recent studies on the topic .
In 2011 , the PubMed website published an article , of which the abstract states : The brain is a highly compartmentalized organ exceptionally susceptible to accumulation of metabolic errors .
Alzheimer ’ s disease ( AD ) is the most prevalent neurodegenerative @ @ @ @ @ @ @ by regional specificity of neural aberrations associated with higher cognitive functions .
Aluminum ( Al ) is the most abundant neurotoxic metal on earth , widely bioavailable to humans and repeatedly shown to accumulate in AD-susceptible neuronal foci .
In spite of this , the role of Al in AD has been heavily disputed based on the following claims : 1 ) bioavailable Al can not enter the brain in sufficient amounts to cause damage , 2 ) excess Al is efficiently excreted from the body , and 3 ) Al accumulation in @ @ @ @ @ @ @ cause of neuronal loss .
Research , however , reveals that : 1 ) very small amounts of Al are needed to produce neurotoxicity and this criterion is satisfied through dietary Al intake , 2 ) Al sequesters different transport mechanisms to actively traverse brain barriers , 3 ) incremental acquisition of small amounts of Al over a lifetime favors its selective accumulation in brain tissues , and 4 ) since 1911 , experimental evidence has repeatedly demonstrated that chronic Al intoxication reproduces neuropathological hallmarks of AD .
Misconceptions about Al bioavailability may @ @ @ @ @ @ @ Al in the pathogenesis of AD .
The hypothesis that Al significantly contributes to AD is built upon very solid experimental evidence and should not be dismissed .
Immediate steps should be taken to lessen human exposure to Al , which may be the single most aggravating and avoidable factor related to AD .
( Emphasis added ) [ 1 ] In March 2018 , the website alzdiscovery published an article , and I quote parts of it here : - Aluminum is an element abundantly present in the earth .
It occurs @ @ @ @ @ @ @ widely used in products ranging from cans and cookware to medications and cosmetics .
Some observational studies suggested a link between brain levels of aluminum and Alzheimer ’ s disease .
Since the association was found , many studies have investigated whether aluminum increases the risk for Alzheimer ’ s .
The findings are far from clear .
Several meta-analyses examined the association between aluminum levels in drinking water and dementia risk , and the evidence is mixed and inconclusive .
The only high-quality study involved almost 4,000 older adults in southwest France @ @ @ @ @ @ @ found that levels of aluminum consumption in drinking water in excess of 0.1 mg/day were associated with a doubling of dementia risk and a 3-fold increase in Alzheimer ’ s risk .
For reference , the 2016 NYC Drinking Water Quality Report states that the concentration of aluminum in NYC drinking water ranged from 0.006-0.057 mg/liter ( average , 0.02 mg/liter ) .
Of the remaining 13 moderate quality studies , 6 found an association between higher aluminum levels in drinking water and increased dementia risk , 4 found no associations , and @ @ @ @ @ @ @ soil levels of aluminum .
Some antacids contain high levels of aluminum , as aluminum hydroxide reduces stomach acidity .
Among medications , antacids and anti-ulceratives contain the highest levels of aluminum… .
A very large meta-analysis of 9 observational studies including more than 6,000 people reported that regular antacid use was not associated with Alzheimer ’ s disease .
Even when the analysis was confined to people who used antacids regularly for over 6 months , there was no association with Alzheimer ’ s .
Studies show that aluminum salts in antiperspirants @ @ @ @ @ @ @ and the little that is absorbed is flushed out by the kidneys .
However , if you regularly shave with a razor , aluminum may be more readily absorbed via small nicks and abrasions .
A meta-analysis of 3 observational studies including more than 1,000 people reported that occupational aluminum dust exposure was not associated with Alzheimer ’ s .
However , further studies that precisely ascertain aluminum exposure are needed .
In a more recent 2016 meta-analysis including 4 studies , the relationship between aluminum exposure and dementia was mixed due to @ @ @ @ @ @ @ article concludes by saying : - It is unclear whether this was intended to mean limiting excessive exposure to aluminium , or limit the distribution of the article to avoid yet another panic reaction from the public .
In an undated article on the Alzheimer ’ s Society website , it mentions metals such as copper , zinc , iron and aluminium , but for the purposes of this article , we are only looking at aluminium .
The article says in part : - At present , there is no strong evidence @ @ @ @ @ @ @ to contact with metals through using equipment or through food or water increases your risk of developing Alzheimer ’ s disease .
However , there are many other metals that are present naturally in the brain .
The current research shows that there is likely to be a relationship between naturally-occurring metals and the development or progression of Alzheimer ’ s disease .
But the evidence doesn ’ t yet show whether this relationship actually causes Alzheimer ’ s disease .
( Emphasis added ) It is also unclear whether reducing metals in @ @ @ @ @ @ @ exposure would have any effect .
These metals are essential to the healthy function of our brain , so further research into changes before or during disease development is also necessary to understand if reducing the amount in the brain would actually be beneficial .
Along with these essential metals , there are other metals that we are exposed to through things such as food .
The body is able to tolerate these metals in small amounts by clearing through the kidneys .
These include aluminium and lead , for example it has @ @ @ @ @ @ @ taken out by the kidneys through organ failure or by exposure to extremely high doses these metals are able to deposit in the brain .
These metals are known to cause negative effects in the brain and have been implicated in several neurological conditions .
It should be noted that the article does not say which neurological conditions .
In 1965 , researchers found that rabbits injected with an extremely high dose of aluminium developed toxic tau tangles in their brains .
This led to speculation that aluminium from cans , cookware , @ @ @ @ @ @ @ could be causing dementia… Importantly , these results were only seen with extremely high exposures that far exceed the levels that can enter the body through food or potentially through contact with aluminium cookware .
( Emphasis added ) Since this study was reported , much research has been done on the relationship of aluminium and Alzheimer ’ s disease .
As yet no study or group of studies has been able to confirm that aluminium is involved in the development of Alzheimer ’ s disease .
Aluminium is seen in the normal @ @ @ @ @ @ @ clear how aluminium is getting into the brain from the blood .
The levels currently seen in peoples brains hasn ’ t been shown to be toxic but an ageing brain may be less able to process the aluminium .
Although aluminium has been seen in amyloid plaques there is no solid evidence that aluminium is increased in the brains of people with Alzheimer ’ s disease .
No convincing relationship between amount of exposure or aluminium in the body and the development of Alzheimer ’ s disease has been established .
( @ @ @ @ @ @ @ drink is in a form that is not easily absorbed in to the body .
Hence the amount taken up is less than 1 % of the amount present in food and drink .
Most of the aluminium taken into the body is cleaned out by the kidneys .
Studies of people who were treated with contaminated dialysis have shown an increase in the amount of aluminium in the brain .
This was believed to be as a result of inadequately monitored dialysis which then led to encephalopathy related dementia .
Methods of @ @ @ @ @ @ @ are better able to predict and prevent this form of dementia .
One large recent study did find a potential role for high dose aluminium in drinking water in progressing Alzheimer ’ s disease for people who already have the disease .
( Author ’ s note : there is no link in the study to this article ) However , multiple other small and large scale studies have failed to find a convincing causal association between aluminium exposure in humans and Alzheimer ’ s disease .
( Emphasis added ) [ 3 @ @ @ @ @ @ @ Online website carried an article claiming aluminium does cause Alzheimer ’ s disease .
I quote : - A link between aluminium and Alzheimer ’ s disease has long existed .
But many scientists say there is not enough evidence to blame the metal , used by thousands for everyday purposes to cook and store food .
However , Professor Chris Exley , from Keele University , says his latest research confirms it does indeed play a role in cognitive decline .
There has been a strong link between human exposure to aluminium @ @ @ @ @ @ @ disease for half a century or more .
However , without definite proof , there is still no consensus in the scientific community about the role of this known neurotoxin in this devastating brain disease .
The latest research from my group , published in the Journal of Trace Elements in Medicine and Biology , makes this link even more compelling .
In my view , the findings are unequivocal in their confirmation of a role for aluminium in some if not all Alzheimer ’ s disease .
At the very least , @ @ @ @ @ @ @ even those who have steadfastly maintained that aluminium has no role in the disease to think again .
( Emphasis added ) I don ’ t believe that is the only factor , but I think it is an important one which should be considered very seriously .
When our new results are put into the context of what is already known about aluminium and Alzheimer ’ s disease their significance becomes overwhelming and compelling .
We already know that the aluminium content of brain tissue in late-onset or sporadic Alzheimer ’ s @ @ @ @ @ @ @ in age-matched controls .
So , individuals who develop Alzheimer ’ s disease in their late sixties and older also accumulate more aluminium in their brain tissue than individuals of the same age without the disease .
Even higher levels of aluminium have been found in the brains of individuals , diagnosed with an early-onset form of sporadic Alzheimer ’ s disease , who have experienced an unusually high exposure to aluminium through the environment or through their workplace .
This means that Alzheimer ’ s disease has a much earlier age of @ @ @ @ @ @ @ early sixties , in individuals who have been exposed to unusually high levels of aluminium in their everyday lives .
We now show that some of the highest levels of aluminium ever measured in human brain tissue are found in individuals who have died with a diagnosis of familial Alzheimer ’ s disease .
The levels of aluminium in brain tissue from individuals with familial Alzheimer ’ s disease are similar to those recorded in individuals who died of an aluminium-induced encephalopathy while undergoing renal dialysis .
In support of our quantitative data @ @ @ @ @ @ @ developed and fully validated method of fluorescence microscopy to provide stunning and unequivocal images of aluminium in brain tissue from familial Alzheimer ’ s disease donors .
Familial Alzheimer ’ s disease is an early-onset form of the disease with first symptoms occurring as early as 30 or 40 years of age .
It is extremely rare , perhaps 2-3 per cent of all cases of Alzheimer ’ s disease .
Its bases are genetic mutations associated with a protein called amyloid-beta , a protein which has been heavily linked with the cause @ @ @ @ @ @ @ disease .
Individuals with familial Alzheimer ’ s disease produce more amyloid beta and the onset of the symptoms of Alzheimer ’ s disease are much earlier in life .
This new research may suggest that these genetic predispositions to early onset Alzheimer ’ s disease are linked in some way to the accumulation of aluminium in brain tissue .
( Emphasis added ) Ageing is the main risk factor for Alzheimer ’ s disease and aluminium accumulates in human brain tissue with ageing .
Environmental or occupational exposure to aluminium results in @ @ @ @ @ @ @ tissue and an early onset form of sporadic Alzheimer ’ s disease .
The genetic predispositions which are used to define familial or early-onset Alzheimer ’ s disease also predispose individuals to higher levels of brain aluminium at a much younger age .
Aluminium is accepted as a known neurotoxin , for example being the cause of dialysis encephalopathy , and its accumulation in human brain tissue at any age can only contribute to any ongoing disease state or toxicity .
We should take all possible precautions to reduce the accumulation of aluminium @ @ @ @ @ @ @ activities and we should start to do this as early in our lives as possible .
[ 4 ] It should be noted the article headline states there is proof , while the article itself says there may be a connection .
In August 2018 , the Canadian Alzheimer ’ s Society website carried an article from which I took these quotes : - We usually think of aluminum as a light silvery metal used to make pots and pans , airplanes or tools , but it also has a non-metallic form .
@ @ @ @ @ @ @ per cent of the earth ’ s surface .
Aluminum in small amounts is referred to as ” trace elements , “ and are found in our environment and in our bodies .
These amounts are “ normal ” and are not harmful .
Because these trace elements are present in the earth , they occur naturally in the foods we eat , in our drinking water and are even added to the water treatment process in some municipalities .
Trace elements of aluminum may also be found in : Cosmetics and personal @ @ @ @ @ @ @ nasal sprays Some drugs in order to make them more effective or less irritating The air we breathe from dry soil , cigarette smoke , pesticide sprays and aluminum-based paint .
Aluminum is present in the body , but its role is not fully understood .
Very little of the aluminum taken in by a healthy individual is actually absorbed ; most of it is flushed out by the kidneys .
( Emphasis added ) Aluminum has been studied for over 40 years as a substance that might be linked to dementia .
@ @ @ @ @ @ @ findings .
Some studies show increased levels of trace elements of aluminum in the brains of people with dementia , while others do not .
Studies have not found an increased incidence of dementia in people with occupational exposure to aluminum .
( Emphasis added ) Tea is one of the few plants whose leaves accumulate larger trace element amounts of aluminum that can seep into the brewed beverage .
However , there is no evidence that dementia is more prevalent in cultures that typically drink large amounts of tea .
( Emphasis @ @ @ @ @ @ @ focused on one animal that is particularly susceptible to aluminum poisoning , which has led to incorrect conclusions about the general effects of aluminum on the body .
( Author ’ s note : which animal is referred to is not mentioned in the article ) It would be difficult to significantly reduce exposure to aluminum simply by avoiding the use of aluminum cookware , foil , beverage cans and other products .
Use of aluminum in pots and pans only contributes to a very small percentage of the average person ’ s @ @ @ @ @ @ @ no convincing evidence that exposure to trace elements of aluminum is connected to the development of dementia .
[ 5 ] An undated article on the Dementia Australia website comes to much the same conclusions .
[ 6 ] While a June 2019 article on the USNews website says much the same .
[ 7 ] Many kitchen pots and pans are made of aluminium , and with the inevitable scraping by utensils , most people will have ingested small quantities of aluminium .
Whether this has a cumulative effect appears inconclusive .
@ @ @ @ @ @ @ railway locomotives , and during that I have come into contact with aluminium electrical conduits that needed repair or replacement .
This involved cutting , grinding and threading of conduit , so I will have breathed in aluminium dust .
Whether this will have any adverse effect on my future mental health only time will tell .
The best I think we can say at the moment is while there is what might termed ‘ circumstantial evidence ’ , there appears to be nothing that can be proven at this point .
At @ @ @ @ @ @ @ apace of the latest research .
If readers have other useful sources , please feel free to share in the comments section below .
About the author : Andy Rowlands is a British independent researcher and writer and assistant editor of the game-changing new climate science book , The Sky Dragon Slayers : Victory Lap .
PRINCIPIA SCIENTIFIC INTERNATIONAL , legally registered in the UK as a company incorporated for charitable purposes .
Head Office : 27 Old Gloucester Street , London WC1N 3AX .
Please DONATE TODAY To Help Our Non-Profit Mission @ @ @ @ @ @

Does Aluminum Contamination Cause Alzheimer’s Disease?
Written by Andy Rowlands
There is speculation of a link between higher levels of aluminum (aluminium) in our bodies and an increased risk of Alzheimer’s Disease. Note the American spelling of aluminium in these articles, which they pronounce ‘aloominum’. Below we cover some of the more recent studies on the topic.
In 2011, the PubMed website published an article, of which the abstract states:
The brain is a highly compartmentalized organ exceptionally susceptible to accumulation of metabolic errors. Alzheimer’s disease (AD) is the most prevalent neurodegenerative disease of the elderly and is characterized by regional specificity of neural aberrations associated with higher cognitive functions. Aluminum (Al) is the most abundant neurotoxic metal on earth, widely bioavailable to humans and repeatedly shown to accumulate in AD-susceptible neuronal foci. In spite of this, the role of Al in AD has been heavily disputed based on the following claims: 1) bioavailable Al cannot enter the brain in sufficient amounts to cause damage, 2) excess Al is efficiently excreted from the body, and 3) Al accumulation in neurons is a consequence rather than a cause of neuronal loss. Research, however, reveals that: 1) very small amounts of Al are needed to produce neurotoxicity and this criterion is satisfied through dietary Al intake, 2) Al sequesters different transport mechanisms to actively traverse brain barriers, 3) incremental acquisition of small amounts of Al over a lifetime favors its selective accumulation in brain tissues, and 4) since 1911, experimental evidence has repeatedly demonstrated that chronic Al intoxication reproduces neuropathological hallmarks of AD. Misconceptions about Al bioavailability may have misled scientists regarding the significance of Al in the pathogenesis of AD. The hypothesis that Al significantly contributes to AD is built upon very solid experimental evidence and should not be dismissed. Immediate steps should be taken to lessen human exposure to Al, which may be the single most aggravating and avoidable factor related to AD. (Emphasis added) [1]
In March 2018, the website alzdiscovery published an article, and I quote parts of it here:-
Aluminum is an element abundantly present in the earth. It occurs naturally in food and water and is widely used in products ranging from cans and cookware to medications and cosmetics. Some observational studies suggested a link between brain levels of aluminum and Alzheimer’s disease. Since the association was found, many studies have investigated whether aluminum increases the risk for Alzheimer’s. The findings are far from clear. Several meta-analyses examined the association between aluminum levels in drinking water and dementia risk, and the evidence is mixed and inconclusive. The only high-quality study involved almost 4,000 older adults in southwest France (the PAQUID study). It found that levels of aluminum consumption in drinking water in excess of 0.1 mg/day were associated with a doubling of dementia risk and a 3-fold increase in Alzheimer’s risk. For reference, the 2016 NYC Drinking Water Quality Report states that the concentration of aluminum in NYC drinking water ranged from 0.006-0.057 mg/liter (average, 0.02 mg/liter). Of the remaining 13 moderate quality studies, 6 found an association between higher aluminum levels in drinking water and increased dementia risk, 4 found no associations, and 1 found a protective effect of higher soil levels of aluminum. Some antacids contain high levels of aluminum, as aluminum hydroxide reduces stomach acidity. Among medications, antacids and anti-ulceratives contain the highest levels of aluminum…. A very large meta-analysis of 9 observational studies including more than 6,000 people reported that regular antacid use was not associated with Alzheimer’s disease. Even when the analysis was confined to people who used antacids regularly for over 6 months, there was no association with Alzheimer’s. Studies show that aluminum salts in antiperspirants are poorly absorbed by the body, and the little that is absorbed is flushed out by the kidneys. However, if you regularly shave with a razor, aluminum may be more readily absorbed via small nicks and abrasions. A meta-analysis of 3 observational studies including more than 1,000 people reported that occupational aluminum dust exposure was not associated with Alzheimer’s. However, further studies that precisely ascertain aluminum exposure are needed. In a more recent 2016 meta-analysis including 4 studies, the relationship between aluminum exposure and dementia was mixed due to the studies being too small.
The article concludes by saying:-
There is no consistent or compelling evidence to associate aluminum with Alzheimer’s disease. Although a few studies have found associations between aluminum levels and Alzheimer’s risk, many others found no such associations. Due to the inconclusive nature of the findings, it may be advisable to limit excessive exposure. (Emphasis added) [2]
It is unclear whether this was intended to mean limiting excessive exposure to aluminium, or limit the distribution of the article to avoid yet another panic reaction from the public.
In an undated article on the Alzheimer’s Society website, it mentions metals such as copper, zinc, iron and aluminium, but for the purposes of this article, we are only looking at aluminium. The article says in part:-
At present, there is no strong evidence to support the fears that coming in to contact with metals through using equipment or through food or water increases your risk of developing Alzheimer’s disease. However, there are many other metals that are present naturally in the brain. The current research shows that there is likely to be a relationship between naturally-occurring metals and the development or progression of Alzheimer’s disease. But the evidence doesn’t yet show whether this relationship actually causes Alzheimer’s disease. (Emphasis added) It is also unclear whether reducing metals in the brain via drugs or reducing our exposure would have any effect. These metals are essential to the healthy function of our brain, so further research into changes before or during disease development is also necessary to understand if reducing the amount in the brain would actually be beneficial. Along with these essential metals, there are other metals that we are exposed to through things such as food. The body is able to tolerate these metals in small amounts by clearing through the kidneys. These include aluminium and lead, for example it has been shown that if they are not taken out by the kidneys through organ failure or by exposure to extremely high doses these metals are able to deposit in the brain. These metals are known to cause negative effects in the brain and have been implicated in several neurological conditions.
It should be noted that the article does not say which neurological conditions.
It continues:-
In 1965, researchers found that rabbits injected with an extremely high dose of aluminium developed toxic tau tangles in their brains. This led to speculation that aluminium from cans, cookware, processed foods and even the water supply could be causing dementia… Importantly, these results were only seen with extremely high exposures that far exceed the levels that can enter the body through food or potentially through contact with aluminium cookware. (Emphasis added) Since this study was reported, much research has been done on the relationship of aluminium and Alzheimer’s disease. As yet no study or group of studies has been able to confirm that aluminium is involved in the development of Alzheimer’s disease. Aluminium is seen in the normal, healthy brain. It is not clear how aluminium is getting into the brain from the blood. The levels currently seen in peoples brains hasn’t been shown to be toxic but an ageing brain may be less able to process the aluminium. Although aluminium has been seen in amyloid plaques there is no solid evidence that aluminium is increased in the brains of people with Alzheimer’s disease. No convincing relationship between amount of exposure or aluminium in the body and the development of Alzheimer’s disease has been established. (Emphasis added) Aluminium in food and drink is in a form that is not easily absorbed in to the body. Hence the amount taken up is less than 1{154653b9ea5f83bbbf00f55de12e21cba2da5b4b158a426ee0e27ae0c1b44117} of the amount present in food and drink. Most of the aluminium taken into the body is cleaned out by the kidneys. Studies of people who were treated with contaminated dialysis have shown an increase in the amount of aluminium in the brain. This was believed to be as a result of inadequately monitored dialysis which then led to encephalopathy related dementia. Methods of dialysis have since been improved and doctors are better able to predict and prevent this form of dementia. One large recent study did find a potential role for high dose aluminium in drinking water in progressing Alzheimer’s disease for people who already have the disease. (Author’s note: there is no link in the study to this article) However, multiple other small and large scale studies have failed to find a convincing causal association between aluminium exposure in humans and Alzheimer’s disease. (Emphasis added) [3]
In December 2016, the Mail Online website carried an article claiming aluminium does cause Alzheimer’s disease. I quote:-
A link between aluminium and Alzheimer’s disease has long existed. But many scientists say there is not enough evidence to blame the metal, used by thousands for everyday purposes to cook and store food. However, Professor Chris Exley, from Keele University, says his latest research confirms it does indeed play a role in cognitive decline. There has been a strong link between human exposure to aluminium and the incidence of Alzheimer’s disease for half a century or more. However, without definite proof, there is still no consensus in the scientific community about the role of this known neurotoxin in this devastating brain disease. The latest research from my group, published in the Journal of Trace Elements in Medicine and Biology, makes this link even more compelling. In my view, the findings are unequivocal in their confirmation of a role for aluminium in some if not all Alzheimer’s disease. At the very least, these new results should encourage everyone and even those who have steadfastly maintained that aluminium has no role in the disease to think again. (Emphasis added) I don’t believe that is the only factor, but I think it is an important one which should be considered very seriously. When our new results are put into the context of what is already known about aluminium and Alzheimer’s disease their significance becomes overwhelming and compelling. We already know that the aluminium content of brain tissue in late-onset or sporadic Alzheimer’s disease is significantly higher than is found in age-matched controls. So, individuals who develop Alzheimer’s disease in their late sixties and older also accumulate more aluminium in their brain tissue than individuals of the same age without the disease. Even higher levels of aluminium have been found in the brains of individuals, diagnosed with an early-onset form of sporadic Alzheimer’s disease, who have experienced an unusually high exposure to aluminium through the environment or through their workplace. (Author’s note: no link to this research is provided in the article) This means that Alzheimer’s disease has a much earlier age of onset, for example, fifties or early sixties, in individuals who have been exposed to unusually high levels of aluminium in their everyday lives. We now show that some of the highest levels of aluminium ever measured in human brain tissue are found in individuals who have died with a diagnosis of familial Alzheimer’s disease. The levels of aluminium in brain tissue from individuals with familial Alzheimer’s disease are similar to those recorded in individuals who died of an aluminium-induced encephalopathy while undergoing renal dialysis. In support of our quantitative data, we have also used a recently developed and fully validated method of fluorescence microscopy to provide stunning and unequivocal images of aluminium in brain tissue from familial Alzheimer’s disease donors. Familial Alzheimer’s disease is an early-onset form of the disease with first symptoms occurring as early as 30 or 40 years of age. It is extremely rare, perhaps 2-3 per cent of all cases of Alzheimer’s disease. Its bases are genetic mutations associated with a protein called amyloid-beta, a protein which has been heavily linked with the cause of all forms of Alzheimer’s disease. Individuals with familial Alzheimer’s disease produce more amyloid beta and the onset of the symptoms of Alzheimer’s disease are much earlier in life. This new research may suggest that these genetic predispositions to early onset Alzheimer’s disease are linked in some way to the accumulation of aluminium in brain tissue. (Emphasis added) Ageing is the main risk factor for Alzheimer’s disease and aluminium accumulates in human brain tissue with ageing. Environmental or occupational exposure to aluminium results in higher levels of aluminium in human brain tissue and an early onset form of sporadic Alzheimer’s disease. The genetic predispositions which are used to define familial or early-onset Alzheimer’s disease also predispose individuals to higher levels of brain aluminium at a much younger age. Aluminium is accepted as a known neurotoxin, for example being the cause of dialysis encephalopathy, and its accumulation in human brain tissue at any age can only contribute to any ongoing disease state or toxicity. We should take all possible precautions to reduce the accumulation of aluminium in our brain tissue through our everyday activities and we should start to do this as early in our lives as possible. [4]
It should be noted the article headline states there is proof, while the article itself says there may be a connection.
In August 2018, the Canadian Alzheimer’s Society website carried an article from which I took these quotes:-
We usually think of aluminum as a light silvery metal used to make pots and pans, airplanes or tools, but it also has a non-metallic form. This form of aluminum makes up eight per cent of the earth’s surface. Aluminum in small amounts is referred to as ”trace elements,“ and are found in our environment and in our bodies. These amounts are “normal” and are not harmful. Because these trace elements are present in the earth, they occur naturally in the foods we eat, in our drinking water and are even added to the water treatment process in some municipalities. Trace elements of aluminum may also be found in: Many processed foods Cosmetics and personal hygiene products, such as deodorants and nasal sprays Some drugs in order to make them more effective or less irritating The air we breathe from dry soil, cigarette smoke, pesticide sprays and aluminum-based paint. Aluminum is present in the body, but its role is not fully understood. Very little of the aluminum taken in by a healthy individual is actually absorbed; most of it is flushed out by the kidneys. (Emphasis added) Aluminum has been studied for over 40 years as a substance that might be linked to dementia. However, there have been many conflicting findings. Some studies show increased levels of trace elements of aluminum in the brains of people with dementia, while others do not. Studies have not found an increased incidence of dementia in people with occupational exposure to aluminum. (Emphasis added) Tea is one of the few plants whose leaves accumulate larger trace element amounts of aluminum that can seep into the brewed beverage. However, there is no evidence that dementia is more prevalent in cultures that typically drink large amounts of tea. (Emphasis added) Unfortunately, earlier animal studies focused on one animal that is particularly susceptible to aluminum poisoning, which has led to incorrect conclusions about the general effects of aluminum on the body. (Author’s note: which animal is referred to is not mentioned in the article) It would be difficult to significantly reduce exposure to aluminum simply by avoiding the use of aluminum cookware, foil, beverage cans and other products. Use of aluminum in pots and pans only contributes to a very small percentage of the average person’s intake of aluminum. Current research provides no convincing evidence that exposure to trace elements of aluminum is connected to the development of dementia. [5]
An undated article on the Dementia Australia website comes to much the same conclusions. [6]
While a June 2019 article on the USNews website says much the same. [7]
Many kitchen pots and pans are made of aluminium, and with the inevitable scraping by utensils, most people will have ingested small quantities of aluminium. Whether this has a cumulative effect appears inconclusive.
One of my hobbies is restoring old railway locomotives, and during that I have come into contact with aluminium electrical conduits that needed repair or replacement. This involved cutting, grinding and threading of conduit, so I will have breathed in aluminium dust. Whether this will have any adverse effect on my future mental health only time will tell.
The best I think we can say at the moment is while there is what might termed ‘circumstantial evidence’, there appears to be nothing that can be proven at this point.
At Principia Scientific International we endeavor to keep apace of the latest research. If readers have other useful sources, please feel free to share in the comments section below.
[1] see: https://pubmed.ncbi.nlm.nih.gov/21157018/
[2] see: https://www.alzdiscovery.org/cognitive-vitality/blog/is-there-a-link-between-aluminum-and-alzheimers
[3] see: https://www.alzheimers.org.uk/about-dementia/risk-factors-and-prevention/metals-and-dementia
[4] see: https://www.dailymail.co.uk/health/article-4047426/Aluminium-DOES-cause-Alzheimer-s-Expert-says-new-findings-confirm-metal-devastating-brain-disease.html
[5] see: https://alzheimer.ca/en/Home/About-dementia/Alzheimer-s-disease/Risk-factors/Aluminum
[6] https://www.dementia.org.au/files/helpsheets/Helpsheet-DementiaQandA19-Aluminium_english.pdf
[7] https://health.usnews.com/conditions/alzheimers/articles/is-there-a-connection-between-aluminum-and-alzheimers-disease
About the author: Andy Rowlands is a regular contributor to Principia Scientific International; a British independent researcher and writer and assistant editor of the game-changing new climate science book, The Sky Dragon Slayers: Victory Lap.
PRINCIPIA SCIENTIFIC INTERNATIONAL, legally registered in the UK as a company incorporated for charitable purposes. Head Office: 27 Old Gloucester Street, London WC1N 3AX.
Please DONATE TODAY To Help Our Non-Profit Mission To Defend The Scientific Method.
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